骨骼肌 SCIE

期刊简介预计审稿时间： 12 Weeks

The only open access journal in its field, Skeletal Muscle publishes novel, cutting-edge research and technological advancements that investigate the molecular mechanisms underlying the biology of skeletal muscle. Reflecting the breadth of research in this area, the journal welcomes manuscripts about the development, metabolism, the regulation of mass and function, aging, degeneration, dystrophy and regeneration of skeletal muscle, with an emphasis on understanding adult skeletal muscle, its maintenance, and its interactions with non-muscle cell types and regulatory modulators.

Main areas of interest include:

-differentiation of skeletal muscle-

atrophy and hypertrophy of skeletal muscle-

aging of skeletal muscle-

regeneration and degeneration of skeletal muscle-

biology of satellite and satellite-like cells-

dystrophic degeneration of skeletal muscle-

energy and glucose homeostasis in skeletal muscle-

non-dystrophic genetic diseases of skeletal muscle, such as Spinal Muscular Atrophy and myopathies-

maintenance of neuromuscular junctions-

roles of ryanodine receptors and calcium signaling in skeletal muscle-

roles of nuclear receptors in skeletal muscle-

roles of GPCRs and GPCR signaling in skeletal muscle-

other relevant aspects of skeletal muscle biology.

In addition, articles on translational clinical studies that address molecular and cellular mechanisms of skeletal muscle will be published. Case reports are also encouraged for submission.

Skeletal Muscle reflects the breadth of research on skeletal muscle and bridges gaps between diverse areas of science for example cardiac cell biology and neurobiology, which share common features with respect to cell differentiation, excitatory membranes, cell-cell communication, and maintenance. Suitable articles are model and mechanism-driven, and apply statistical principles where appropriate; purely descriptive studies are of lesser interest.

《骨骼肌》是该领域唯一的开放获取期刊，发表研究骨骼肌生物学背后分子机制的新颖、尖端研究和技术进步。为反映该领域研究的广度，该期刊欢迎有关骨骼肌的发育、代谢、质量和功能调节、衰老、退化、营养不良和再生的稿件，重点是了解成人骨骼肌、其维持及其与非肌肉细胞类型和调节剂的相互作用。

主要感兴趣的领域包括：

-骨骼肌分化-

骨骼肌萎缩和肥大-

骨骼肌衰老-

骨骼肌再生和退化-

卫星细胞和卫星样细胞的生物学-

骨骼肌营养不良性退化-

骨骼肌中的能量和葡萄糖稳态-

非营养不良性遗传疾病骨骼肌，如脊髓性肌萎缩和肌病-

神经肌肉接头的维持-

瑞安诺丁受体和钙信号在骨骼肌中的作用-

核受体在骨骼肌中的作用-

GPCR 和 GPCR 信号在骨骼肌中的作用-

骨骼肌生物学的其他相关方面。

此外，还将发表关于骨骼肌分子和细胞机制的转化临床研究文章。也鼓励提交病例报告。

骨骼肌反映了骨骼肌研究的广度，并弥合了不同科学领域之间的差距，例如心脏细胞生物学和神经生物学，它们在细胞分化、兴奋膜、细胞间通讯和维持方面具有共同特征。合适的文章是模型和机制驱动的，并在适当的地方应用统计原理；纯粹的描述性研究不太有趣。

《Skeletal Muscle》(骨骼肌)编辑部通讯方式为CAMPUS, 4 CRINAN ST, LONDON, ENGLAND, N1 9XW。如果您需要协助投稿或润稿服务，您可以咨询我们的客服老师。我们专注于期刊投稿服务十年，熟悉发表政策，可为您提供一对一投稿指导，避免您在投稿时频繁碰壁，节省您的宝贵时间，有效提升发表机率，确保SCI检索（检索不了全额退款）。我们视信誉为生命，多方面确保文章安全保密，在任何情况下都不会泄露您的个人信息或稿件内容。